BROWN RECLUSE
Loxosceles Reclusa
Order: Araneae
Sub-Order:
Family: Loxoscelidae
Genus: Loxosceles
Species: Reclusa
Male size: 6 (span 25 mm)
Female size: 20 mm (span 50 mm)
Habitat: Dry areas
Range: Midwest and Southern US
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Identification
The brown recluse belongs to the group of violin spiders because of a fiddle-shaped pattern it
shows on the head region of the dorsal side of the cephalothorax. Its brown body is fairly small
and its legs are long and slender, the dorsal pattern is normally shiny and darker. It's a
relatively hairless spider seemingly smooth. It's possible to mistake it with other common
house spider but its characteristic eye number and arrangement stands it out: it has only 6
eyes arranged in three pairs.
Behaviour
The brown recluse, living up to its name, is naturally non aggressive toward humans and
prefers to live in undisturbed attics, woodpiles, and storage sheds. Its web is quite irregular
and usually located in dark areas, it feeds on insects like crickets but its nutritional practice is
rather singular owing to its incredible capability to survive up to five months with no food or
water. Female deposits its eggs n white silken cases than can reach 10 mm in diameter, after
about 30 days spiderlings emerge and in base of weather condition and food availability they
slowly reach maturity in 12 month on average.
Venom
Brown recluse venom is considered cytotoxic and haemolytic; between its components we find
various enzymes such as hyaluronidase, deoxyribonuclease, ribonuclease, alkaline
phosphatase and lipase, but the one thought to be the principle responsible of toxicity is
sphingomyelinase D.
The bite initially cause a stinging sensation that evolves in severe pain and itching in 6-8
hours, local reaction includes redness of the skin, tenderness and possible blistering; after 12-
24 hours fever, nausea, chills and joint pain occur. Edema develops in the bite area, the tissue
becomes ischemic owing to the acting of necrotic and haemolytic enzymes; then disseminated
intravascular coagulation and renal failure arise.
The intense inflammatory response that involves prostaglandins and chemotactic infiltration of
neutrophils leads to the activation of mediator C-reactive protein and complement that
progressively contribute to the necrotic degeneration.